Feingold diet

Dr Ben F. Feingold, once a Professor of Allergy in San Francisco, claimed that hyperactivity was increasing in proportion to the level of food additives and proposed a specific diet believing that it would help 50% of hyperactive children.

The Feingold diet excluded cola drinks, chocolate, preservatives and flavor additives, as well as salicylates that occur naturally in fruit such as tomatoes, strawberries, pineapples and oranges. However pineapple juice was suggested as a "safe" drink.

The effectiveness of the Feingold diet has been heavily disputed. Most studies have shown that only 5% of children diagnosed with ADHD benefited from the diet. Other studies have shown a figure of 60%.

Vitamin B6

In the 1980s the vitamin B6 promoted as a helpful remedy for children with learning difficulties including inattentiveness. After that, zinc was promoted for ADD and autism. Multivitamins later became the claimed solution. Thus far, no reputable research has appeared to support any of these claims, except in cases of malnutrition.

Neurofeedback

There has been a lot of interesting work done with neurofeedback and ADHD. Children are taught, using video game-like technology, how to control their brain waves. This has a very high success rate, but is not widely used, or covered by insurance. Many professionals consider the treatment promising, but state that there is not yet sufficient evidence that it works after the immediate treatment is complete.

Possible causes

ADHD is broadly defined and pervasive, and the symptoms attributed to ADHD likely have a variety of different causes. The initial triggers could include genetic vulnerabilities, viral or bacterial infections, brain injury, or nutritional deficits. There has been a surge in alternative approaches to ADHD, but these have been vigorously disputed.

Neuro-chemical imbalance

There is increasing evidence that variants in the gene for the dopamine transporter are related to the development of ADHD (Roman et al., 2004, American Journal of Pharmacogenomics 4:83-92). This evidence is consonant with the theory of inefficacy of dopamine in people with ADD/ADHD; according to other recent studies, people with ADHD usually have relatively high dopamine transporter levels, which clears dopamine from between neurons before the full effect is gained from dopamine. Stimulant medications used to treat ADHD are all capable of either inhibiting the action of dopamine transporter (as methylphenidate does) or promoting the release of dopamine itself (as the amphetamine-class medications do). Therefore, it is theorized that stimulant medication allows the brain to enhance the effect of dopamine by blocking dopamine transporters or increasing the release of dopamine. Currently this is the most widely accepted model of ADD/ADHD etiology in the scientific and medical community. New studies consider the possibility that norepinephrine also plays a role.